Michael Sendtner

The paper investigates how CASPR2 autoantibody subclasses, particularly IgG4, affect neuronal excitability and potassium channel function in patients with neuropathic pain.

This paper demonstrates that a novel C-CDNF fragment can protect neurons in Parkinson's and ALS models through non-invasive peripheral delivery.

The paper isolates nerve terminals from NaV1.9 knockout mice and performs proteomic analysis to identify dysregulated pathways in neuropathic pain signaling.

The paper introduces a method to compress genetic data using autoencoders that leverage linkage disequilibrium structure, achieving high reconstruction accuracy while reducing data size.

This study shows that inhibiting hnRNP R reduces axonal tau levels and tau tangles in Alzheimer's disease mice, suggesting a targeted therapeutic approach.

This study investigates stage-dependent molecular and cellular changes in a rat model of hypertension, revealing vascular and neural pathways linked to inflammation and cognitive dysfunction.

The paper investigates dynamic remodeling of endoplasmic reticulum and ribosomes in axons of motoneurons in response to BDNF stimulation, revealing their role in local translation and synapse maintenance.

This study shows CDNF reduces ER stress and improves motor neuron survival in multiple ALS models, independent of genetic cause.

This study shows that an ALS-associated FUS mutation disrupts synaptic vesicle docking and neuromuscular transmission in C. elegans.