Author

Wei Xiang

Friedrich-Alexander-Universität Erlangen-NürnbergORCID 0000-0002-4785-9934Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Wei Xiang4 papers · Friedrich-Alexander-Universität Erlangen-Nürnberg

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:metagenomics· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Aiden HaghikiaiD· 1 shared Arne GessneriD· 1 shared Cezmi A. AkdişiD· 1 shared Daniele MauroiD· 1 shared Eva SchmidiD· 1 shared Fabian SchälteriD· 1 shared Felix BeyeriD· 1 shared Francesco CicciaiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

Aiden HaghikiaORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Gut-specific H3R signaling orchestrates microglia-dependent resolution of peripheral inflammation

Kerstin Dürholz, Leona Ehnes, Mathias Linnerbauer, Eva Schmid, Heike Danzer, Michael Hinzpeter-Schmidt, Lena Lößlein, Lena Amend, Michael Frech, Vugar Azizov, Fabian Schälter, Arne Gessner, Sébastien Lucas, Till-Robin Lesker, Regina Verena Taudte, , Mario M. Zaiss

The paper identifies gut microbiota-derived histamine as a factor enabling the CNS to suppress peripheral inflammation in RA by activating H3R in the intestine and modulating microglia.

Journal of Clinical Investigation · Jul 10, 2025Peer-reviewedImmunology