Venkatesh P. Thirumalaikumar

This study identifies an aphid protein, AAF1, that manipulates plant autophagy to enhance aphid survival and plant growth.

Hopeaphenol inhibits histidine kinase signaling in both bacterial pathogens and plant hosts, reducing virulence and enhancing immunity.

The paper shows that translocon remodeling affects ribosomal frameshifting and viral polyprotein maturation in alphaviruses, reducing viral fitness.

This study identifies new TGM2-catalyzed histaminylation marks on core histones in cancer cells and reveals their crosstalk with γH2AX formation in DNA damage responses.

This study integrates multi-omics data from quinoa to identify genetic factors controlling its metabolome and stress response, revealing key genes involved in metabolite biosynthesis.

This study investigates how juglone affects Arabidopsis thaliana by depleting thiols and disrupting proteostasis, revealing its phytotoxic mechanisms.

This study identifies proteomic changes in Crocosphaera subtropica during light-dark cycles and nitrogen fixation, revealing coordinated regulation of metabolic pathways and nitrogenase expression.

This study shows that oxybutynin rescues proliferative capacity and metabolic function in Complex III-defective mitochondrial progenitor cells.

This study uses multi-omics analysis to investigate heat stress memory in Arabidopsis, identifying temporal transcriptomic, proteomic, and metabolomic responses during recovery.

The paper introduces PROMIS, a co-fractionation mass spectrometry technique integrated with chromatographic methods to map protein-metabolite interactions in E. coli, identifying 994 interactions and revealing novel functional insights.

This study uses snRNA-seq to identify cell-type-specific responses to silicon treatment in soybean leaves, revealing novel Si-induced cell clusters and gene expression changes related to defense mechanisms.

Natural variation at KCS4 underlies differential accumulation of polyunsaturated triacylglycerols under carbon starvation by acting as a regulatory branch point in fatty acid fate.