Author

Diego Gonzalez‐Perez

Universidade de Santiago de CompostelaORCID 0000-0002-5029-2289Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Aurora Gómez-DurániD· 2 shared Alejandro Failde-FiestrasiD· 1 shared Alex Miralles-DomingueziD· 1 shared Celia M. PomboiD· 1 shared Cheng CheniD· 1 shared Clara AlconiD· 1 shared Cristina RiobelloiD· 1 shared Dafni ChondronasiouiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCell Biology

Aurora Gómez-DuránORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

SENOMORPHIC EFFECT OF GENETIC AND CHEMICAL PARTIAL REPROGRAMMING

Victor Nuñez-Quintela, Jérémy Chantrel, Miguel Angel Prados, Pablo Pedrosa, Italo Lorandi, Lucia Lorenzo Rodriguez, Cheng Chen, Raquel Paredes-Paredes, Alejandro Failde-Fiestras, Diego Gonzalez‐Perez, Alex Miralles-Dominguez, Sabela Da Silva‐Álvarez, Ramón Lobato-Busto, Miguel González‐Barcia, Clara Alcon, , Manuel Collado

This study shows genetic and chemical partial reprogramming targets senescent cells to reduce aging-related inflammation and improve mitochondrial function in aged mice without restoring proliferation.

bioRxiv · Jan 13, 2026 · PreprintCell Biology

Rapamycin reverts cavernoma endothelial cell phenotype and when combined with lapatinib ameliorates chronic lesions

Mar García-Colomer, José E Martínez, Luis Antonio Díaz-Gómez, Miriam Sartages, Eva M. Esquinas-Román, Cristina Riobello, David Martinez-Delgado, Diego Gonzalez‐Perez, Aurora Gómez-Durán, Miguel Fidalgo, Marta Varela‐Rey, Celia M. Pombo, Juan B. Zalvide

This study shows rapamycin reverses gene expression in Ccm3-deficient endothelial cells and, when combined with lapatinib, reduces lesion volume in a chronic CCM model.

bioRxiv · Nov 3, 2025 · PreprintCell Biology