Author

Colin Forster

Heidelberg UniversityORCID 0000-0002-5896-1491Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell signaling & trafficking· 1 Organism:human· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Paul RothhaariD· 2 shared Tomke Arand· 2 shared Volker LohmanniD· 2 shared Andreas PichlmairiD· 1 shared Antonio PirasiD· 1 shared Christian HeussiD· 1 shared Christoph Sarrazin· 1 shared Eva HegeriD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersMicrobiology

Paul RothhaarORCID

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2 papers

Recent·Most cited

MicrobiologyPLoS PathogensPeer-reviewedAug 5, 2026

A novel dimerization site in non-structural protein 5A of hepatitis C virus regulates viral replication fitness

Paul Rothhaar, Thibault Tubiana, Colin Forster…·Volker Lohmann

This study identifies a dimerization site in NS5A that enhances HCV replication fitness by promoting self-interaction, which inhibits NS3 and NS5B activity.

DOI 10.1371/journal.ppat.1014239

humanproteomics

𝕏 PostBluesky

MicrobiologybioRxivPreprintFeb 4, 2026

Hepatitis C virus replication fitness as a determinant of antiviral therapy outcome

Ha-Gyu Thomas Seong, Tomke Arand, Colin Forster…·Paul Rothhaar

This study examines how HCV replication fitness affects antiviral treatment outcomes, finding that high replication fitness can compensate for resistance mutations and contribute to DAA treatment failure.

1 citation

experimental assayhost-pathogen & immune response

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