Author

Morgan E. McNellis

Tufts UniversityORCID 0000-0002-6055-262XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Organism:C. elegans· 2 Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Aimee SheniD· 2 shared A. Ioana Stoica· 1 shared Bree B. AldridgeiD· 1 shared Ekaterina E. Zheleznova HeldweiniD· 1 shared Emily R. Forster· 1 shared Eun Seon ChungiD· 1 shared Gonzalo L. González‐Del PinoiD· 1 shared Gregory A. HarrisoniD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersMicrobiology

Aimee ShenORCID

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3 papers

Recent·Most cited

MicrobiologyPLoS BiologyPeer-reviewedFeb 2, 2026

The CspC:CspA heterodimer transduces germinant and co-germinant signals during Clostridioides difficile spore germination.

Morgan E. McNellis, Gonzalo L. González‐Del Pino, Juan A. Serrano-Jiménez…·Aimee Shen

This study identifies the CspC:CspA heterodimer as the signaling complex that transduces germinant and co-germinant signals during C. difficile spore germination.

4 citationstop 1%

C. elegansexperimental assay

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MicrobiologyJournal of BacteriologyPeer-reviewedNov 26, 2025

Functional redundancy between penicillin-binding proteins during asymmetric cell division in Clostridioides difficile

Shailab D. Shrestha, Jules M. Dressler, Gregory A. Harrison…·Aimee Shen

The paper shows that SpoVD's catalytic activity is dispensable for asymmetric division in C. difficile due to PBP3's compensatory role, suggesting functional redundancy among bPBPs.

5 citations

C. elegansproteomics

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MicrobiologybioRxivPreprintJul 14, 2025

Increased proportion of growth-arrested bacilli in acidic pH adaptation promotes Mycobacterium tuberculosis treatment survival

Eun Seon Chung, William C. Johnson, Maliwan Kamkaew…·Bree B. Aldridge

This study shows that Mycobacterium tuberculosis adapts to acidic conditions by increasing growth-arrested cells, which enhance drug tolerance and survival.

DOI 10.1101/2025.07.14.664820

experimental assayhost-pathogen & immune response

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