Author

Robert A. Ellingford

King's College LondonORCID 0000-0002-6455-3013Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:electronic health records· 1 Approach:statistical model· 1

Frequent Co-Authors

M. Albert BassoniD· 2 shared Alberto GalbuseraiD· 1 shared Alessandro GozziiD· 1 shared Alessio DeloguiD· 1 shared Angela CarusoiD· 1 shared Carlo Sala FrigerioiD· 1 shared Caterina MichettiiD· 1 shared Cathy FernandesiD· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersNeuroscience

M. Albert BassonORCID

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3 papers

Recent·Most cited

NeurosciencePLoS BiologyPeer-reviewedJul 23, 2024

Selective suppression of oligodendrocyte-derived amyloid beta rescues neuronal dysfunction in Alzheimer's Disease

Rikesh M. Rajani, Robert A. Ellingford, Mariam Hellmuth…·Marc Aurel Busche

This study shows that oligodendrocytes produce amyloid beta in Alzheimer's Disease and that suppressing this production improves brain pathology and neuronal function.

60 citationstop 1%

mouseexperimental assay

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NeuroscienceMolecular PsychiatryPeer-reviewedApr 9, 2021

Cell-type-specific synaptic imbalance and disrupted homeostatic plasticity in cortical circuits of ASD-associated Chd8 haploinsufficient mice

Robert A. Ellingford, Martyna J. Panasiuk, Emilie Rabeshala de Meritens…·Laura C. Andreae

This study examines synaptic imbalances and homeostatic plasticity disruption in ASD-associated Chd8 haploinsufficient mice, revealing altered excitatory and inhibitory transmission in cortical circuits.

54 citationstop 4%

mouseelectronic health records

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NeuroscienceCerebral CortexPeer-reviewedFeb 27, 2018

Altered neocortical gene expression, brain overgrowth and functional over-connectivity in Chd8 haploinsufficient mice

Philipp Suetterlin, Shaun Hurley, Conor Mohan…·M. Albert Basson

This study examines Chd8 haploinsufficient mice, finding brain overgrowth, altered gene expression, and functional connectivity changes linked to autism-like features.

131 citationstop 1%

mousebulk RNA-seq

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