Author

Julian E. Stelzer

Case Western Reserve UniversityORCID 0000-0002-6870-9054Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biophysics· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1 Organism:mouse· 1 Approach:simulation· 1

Frequent Co-Authors

Alexandra V. SchmidtiD· 1 shared Bertrand C.W. TanneriD· 1 shared Christopher M. YengoiD· 1 shared Eric S. GoetzmaniD· 1 shared John R. KotteriD· 1 shared Jonathan F. WenkiD· 1 shared Kenneth S. CampbelliD· 1 shared Kerry S. McDonaldiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperPhysiology

Alexandra V. SchmidtORCID

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2 papers

Recent·Most cited

Hearts may grow concentrically to balance ATP supply and demand and eccentrically to stabilize titin-based stress

John R. Kotter, Steve Leung, Thomas Kampourakis, Lik-Chuan Lee, Jonathan F. Wenk, Michael J. Moulton, Bertrand C.W. Tanner, Stuart G. Campbell, Christopher M. Yengo, Kerry S. McDonald, Julian E. Stelzer, Kenneth S. Campbell

This paper uses simulations to show that concentric and eccentric cardiac growth are regulated by ATP homeostasis and titin-based stress, respectively, and can model growth patterns from both genetic and clinical causes.

bioRxiv · May 13, 2026 · PreprintBiophysics

Systemic Metabolic Rewiring in a Mouse Model of Left Ventricular Hypertrophy

Alexandra V. Schmidt, Tharika Thambidurai, Olivia D’Annibale, Sivakama S. Bharathi, Timothy C. Wood, Eric S. Goetzman, Julian E. Stelzer

This study investigates systemic metabolic changes in a mouse model of left ventricular hypertrophy, finding that exogenous long-chain fatty acid supplementation improves mitochondrial function but does not fully reverse the disease phenotype.

International Journal of Molecular Sciences · Oct 17, 2025Peer-reviewedPhysiology