Vishwas Mishra

The paper identifies resource-access breadth as a protective trait in non-toxigenic Clostridioides difficile strains, distinguishing protection from low disease potential through metabolic and competitive profiling.

This study shows that aged mice have increased susceptibility to Citrobacter rodentium infection but retain distinct molecular and inflammatory responses depending on the pathogen's virulence.

The paper introduces a platform using Citrobacter rodentium to generate antibodies against bacterial polysaccharides and proteins without requiring purified or synthetic antigens.

This study shows that colonic ILC2s are activated during Citrobacter rodentium infection, secreting type 2 cytokines, and that IL-18 drives their activation.

This study shows that resolving an enteric infection with Citrobacter rodentium protects mice from chemically-induced colitis via IL-17A-dependent immune reprogramming.

The study identifies 12 accessory T3SS effectors in Citrobacter rodentium that influence infection outcomes without affecting virulence.