Author

Getachew Debas Belew

Ohio UniversityORCID 0000-0002-7338-1938Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

Agnes O PortuphyiD· 2 shared Raziyeh Abdolahipour· 2 shared Sivarajan KumarasamyiD· 2 shared Sobia ZaidiiD· 2 shared Sonia Michael NajjariD· 2 shared Abhishek P ShresthaiD· 1 shared Ali ZarrinpariD· 1 shared Amira F. GoharaiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersPhysiology

Agnes O PortuphyORCID

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2 papers

Recent·Most cited

Defective insulin clearance plays a primary role in the pathogenesis of chronic kidney disease in mice with null deletion of Ceacam2 gene

Sivarajan Kumarasamy, Getachew Debas Belew, Agnes O Portuphy, Raziyeh Abdolahipour, Sobia Zaidi, Haixin Yang, Lucia Russo, Simona S. Ghanem, Amira F. Gohara, Ramiro Malgor, Sonia Michael Najjar

This study shows that loss of CEACAM2 in mice impairs renal insulin clearance, leading to hyperinsulinemia, insulin resistance, and kidney dysfunction.

bioRxiv · Nov 23, 2025 · PreprintPhysiology

Conditional deletion of CEACAM1 causes hepatic stellate cell activation

Harrison T. Muturi, Hilda E. Ghadieh, Suman Asalla, Sumona Ghosh Lester, Getachew Debas Belew, Sobia Zaidi, Raziyeh Abdolahipour, Abhishek P Shrestha, Agnes O Portuphy, Hannah L. Stankus, Raghd Abu Helal, Stefaan Verhulst, Sérgio M. Duarte, Ali Zarrinpar, Leo A. van Grunsven, , Sonia Michael Najjar

This study shows that CEACAM1 loss in hepatic stellate cells (HSCs) activates them, leading to myofibroblastic transformation and fibrosis without insulin resistance or steatosis.

Molecular Metabolism · Aug 19, 2024Peer-reviewedPhysiology