Author

Enes Cicek

Koç UniversityORCID 0000-0002-7452-2253Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Enes Cicek1 paper · Jena University Hospital

Biology Disciplines

Click to filter author papers

Cancer Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Assay:metabolomics· 1

Frequent Co-Authors

Batuhan Mert KalkaniD· 2 shared Beste KanevetciiD· 2 shared Ceyda AçılaniD· 2 shared Selahattin Can ÖzcaniD· 2 shared Evrim GokseliD· 1 shared Nazlı Ezgi ÖzkaniD· 1 shared Nurhan ÖzlüiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

7 top collaborators · 19 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

Batuhan Mert KalkanORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cancer BiologyCell Communication and SignalingPeer-reviewedApr 7, 2026

Stress adaptation pathways and HA-CD44 signaling maintain the survival of pancreatic cancer cells with centrosome amplification

Selahattin Can Özcan, Evrim Goksel, Batuhan Mert Kalkan…·Ceyda Açılan

This study identifies metabolic and signaling pathways that enable pancreatic cancer cells with centrosome amplification to survive stress, highlighting vulnerabilities for therapeutic targeting.

1 citation

humanproteomics

𝕏 PostBluesky

Cancer BiologybioRxivPreprintJan 27, 2026

Proximity labeling reveals cell cycle-specific NEK2 interactions and a regulatory axis controlling NUSAP1 stability

Enes Cicek, Selahattin Can Özcan, Beste Kanevetci…·Ceyda Açılan

The paper uses proximity labeling and mass spectrometry to map NEK2 interactions across the cell cycle, revealing a NEK2-NUSAP1 degradation pathway.

DOI 10.64898/2026.01.25.701545

humanproteomics

𝕏 PostBluesky