Henrik Bringmann

This study shows that C. elegans distinguishes pathogen proximity through compound types, activating EV-dependent physiological responses tailored to immediate or anticipated threats.

This study shows that the RIS neuron in C. elegans can enhance survival even when sleep behavior is disrupted, suggesting survival and sleep are separable functions of sleep neurons.

The paper introduces OptoGenBox, a device for long-term optogenetic manipulation of C. elegans, showing it reduces survival when increasing arousal or sleep deprivation.

The paper identifies wake-active neurons that depolarize sleep-active neurons to initiate sleep in C. elegans by coordinating locomotion circuits during lethargus.

The paper identifies EGFR signaling in the RIS neuron and its indirect activation via the ALA neuron as mechanisms for sleep induction following cellular stress in C. elegans.