Author

Rosanna C. T. Wright

University of ManchesterORCID 0000-0002-8095-8256Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:bacteria (other)· 3 Approach:experimental assay· 3 Research Focus:gene regulatory network inference· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

Michael A. BrockhurstiD· 2 shared Anna Richmond· 1 shared Aras KadiogluiD· 1 shared Christian HarrisoniD· 1 shared Damian William RivettiD· 1 shared Danna R. GiffordiD· 1 shared Edze Rients WestraiD· 1 shared Eleri A. Ashworth· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersMicrobiology

Michael A. BrockhurstORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The pQBR mercury resistance plasmids: a model set of sympatric environmental mobile genetic elements

Victoria T Orr, Ellie Harrison, Damian William Rivett, Rosanna C. T. Wright, James P. J. Hall

This study analyzes 26 mercury resistance plasmids from Pseudomonas spp., revealing their genetic diversity, transposon-driven evolution, and ability to mobilize chromosomal traits.

Microbial Genomics · Jul 22, 2026Peer-reviewedMicrobiology

Resistance mutation supply modulates the benefit of CRISPR immunity against virulent phages

Rosanna C. T. Wright, Simon C. Lovell, Anna Richmond, Christian Harrison, Eleri A. Ashworth, Aras Kadioglu, Jo Fothergill, Ville‐Petri Friman, Edze Rients Westra, Michael A. Brockhurst

The paper shows CRISPR immunity in Pseudomonas aeruginosa varies in fitness benefit against virulent phages, depending on the likelihood of evolving receptor-based resistance through spontaneous mutations.

bioRxiv · Oct 5, 2025 · PreprintMicrobiology

Inflammation-like environments limit the loss of quorum sensing in Pseudomonas aeruginosa

Taoran Fu, Rosanna C. T. Wright, Danna R. Gifford, Knight Cg, Michael A. Brockhurst

The paper shows that host inflammatory environments constrain the loss of quorum sensing in Pseudomonas aeruginosa by linking it to oxidative stress and amino acid availability.

mSystems · Jul 7, 2025Peer-reviewedEvolutionary Biology