Author

Ermei M. Mäkilä

University of TurkuORCID 0000-0002-8300-6533Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Assay:imaging· 1 Approach:microscopy & imaging analysis· 1 Organism:mouse· 1

Frequent Co-Authors

Aleksi IsomursuiD· 1 shared Frédéric FercoqiD· 1 shared Gautier FollainiD· 1 shared Guillaume JacquemetiD· 1 shared Helene Helo· 1 shared James R. W. ConwayiD· 1 shared Joanna W. PylvänäineniD· 1 shared Johanna IvaskaiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

Aleksi IsomursuORCID

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Author profile →

1 paper

Recent·Most cited

Pancreatic cancer cells breach endothelial barriers through protrusion-driven invasion or endothelial retraction

Gautier Follain, Sujan Ghimire, Monika Vaitkevičiūtė, Helene Helo, Joanna W. Pylvänäinen, Frédéric Fercoq, Aleksi Isomursu, Megan R. Chastney, Omkar Joshi, Marco De Donatis, Ermei M. Mäkilä, James R. W. Conway, Marko Salmi, Sara A. Wickström, Leo M. Carlin, , Guillaume Jacquemet

This study identifies two distinct mechanisms by which pancreatic cancer cells extravasate: protrusion-driven invasion and endothelial retraction, with implications for anti-metastatic strategies.

bioRxiv · Sep 3, 2026 · PreprintCell Biology