Author

Omkar Joshi

Åbo Akademi UniversityORCID 0000-0002-7410-9987Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell signaling & trafficking· 3 Organism:human· 3 Assay:proteomics· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Johanna IvaskaiD· 4 shared Gautier FollainiD· 3 shared Megan R. ChastneyiD· 3 shared Guillaume JacquemetiD· 2 shared James R. W. ConwayiD· 2 shared Joanna W. PylvänäineniD· 2 shared Michal DibusiD· 2 shared Sara A. WickströmiD· 2 shared

Activity by Year

2025-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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4 shared papersCell Biology

Johanna IvaskaORCID

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Pancreatic cancer cells breach endothelial barriers through protrusion-driven invasion or endothelial retraction

Gautier Follain, Sujan Ghimire, Monika Vaitkevičiūtė, Helene Helo, Joanna W. Pylvänäinen, Frédéric Fercoq, Aleksi Isomursu, Megan R. Chastney, Omkar Joshi, Marco De Donatis, Ermei M. Mäkilä, James R. W. Conway, Marko Salmi, Sara A. Wickström, Leo M. Carlin, , Guillaume Jacquemet

This study identifies two distinct mechanisms by which pancreatic cancer cells extravasate: protrusion-driven invasion and endothelial retraction, with implications for anti-metastatic strategies.

bioRxiv · Sep 3, 2026 · PreprintCell Biology

Adhesion-derived condensates control component availability to regulate adhesion dynamics

Michal Dibus, Megan R. Chastney, Giray Enkavi, Gautier Follain, Omkar Joshi, Ilpo Vattulainen, Johanna Ivaska

This study shows that TNS1 phase separation regulates adhesion dynamics by controlling the availability of inactive adhesion proteins through phosphorylation-dependent condensate formation.

Nature Communications · Jun 5, 2026Peer-reviewedCell Biology

Restoring mechanophenotype reverts malignant properties of ECM-enriched vocal fold cancer

Jasmin Kaivola, Karolina Punovuori, Megan R. Chastney, Hind Abdo, Gautier Follain, Mathilde Mathieu, Omkar Joshi, Yekaterina A. Miroshnikova, Fabian Krautgasser, Jasmin Di Franco, James R. W. Conway, Sofia Held, Fabien Bertillot, Jaana Hagström, Antti A. Mäkitie, , Johanna Ivaska

This study shows that restoring mechanophenotype or targeting YAP/TAZ can reverse malignant properties of vocal fold cancer by reducing nuclear YAP and β-catenin levels.

Nature Materials · Feb 20, 2026Peer-reviewedCell Biology

Filopodome proteomics identifies CCT8 as a MYO10 interactor critical for filopodia functions

Ana Popović, Neil J. Ball, Mitro Miihkinen, Omkar Joshi, Michal Dibus, Marjaana Ojalill, Joanna W. Pylvänäinen, Johanna Ivaska, Benjamin Thomas Goult, Guillaume Jacquemet

The paper identifies CCT8 as a MYO10 interactor critical for filopodia functions through proteomics and functional studies in cancer cells.

bioRxiv · Dec 5, 2025 · PreprintCell Biology