Author

Binbin Li

Mayo ClinicORCID 0000-0002-8572-8003Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Danielle Marie CarlsoniD· 2 shared Rory L. SmootiD· 2 shared Sumera Irie IlyasiD· 2 shared Akhilesh PandeyiD· 1 shared Amro M. AbdelrahmaniD· 1 shared Aushinie M. Abeynayake· 1 shared Caitlin B. ConboyiD· 1 shared Darren J. BakeriD· 1 shared

Activity by Year

2021-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersCancer Biology

Danielle Marie CarlsonORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJun 25, 2026

Therapy-induced senescent-like cancer cells drive macrophage-mediated immunosuppression in cholangiocarcinoma

Binbin Li, Jingchun Yang, Meina Cai…·Sumera Irie Ilyas

This study identifies therapy-induced senescent-like cancer cells as drivers of immunosuppression in cholangiocarcinoma by promoting macrophage-mediated suppression of CD8+ T cells.

DOI 10.64898/2026.06.24.734341

humanmouse

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Cancer BiologybioRxivPreprintJan 23, 2026

Multiomics-driven discovery of predictive biomarkers and strategies to overcome resistance to SFK-YAP inhibition in cholangiocarcinoma

Hendrien Kuipers, Danielle Marie Carlson, Erik Jessen…·Rory L. Smoot

This study identifies biomarkers and resistance mechanisms to SFK-YAP inhibition in cholangiocarcinoma using multiomic profiling and shows synergy with chemotherapy.

DOI 10.64898/2026.01.21.699926

humanbulk RNA-seq

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Genetic & Genomic MedicinemedRxivPreprintJul 21, 2021

Upregulation of TBX1 by genetic variants are associated with human congenital heart disease

Li-Wei Yu, Binbin Li, Hongyan Wang

This study identifies a genetic variant in TBX1 associated with congenital heart disease and shows it increases TBX1 promoter activity and risk.

DOI 10.1101/2021.07.21.21260948

humanstatistical model

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