Author

María de Haro

Baylor College of MedicineORCID 0000-0002-8654-2526Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:Drosophila· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Ismael Al‐RamahiiD· 2 shared Adulfo Anaya Amador· 1 shared Alma M. PereziD· 1 shared Andrew Laitman· 1 shared Boxun LuiD· 1 shared Dylan Lawrence Timperman· 1 shared Elena K. RuffiD· 1 shared Genevera I. AlleniD· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersNeuroscience

Ismael Al‐RamahiORCID

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2 papers

Recent·Most cited

Modulation of Karyopherin Levels Attenuates Mutant Ataxin-1-Induced Neurodegeneration

Elena K. Ruff, Dylan Lawrence Timperman, Adulfo Anaya Amador, Isabella Aguirre‐Lamus, Khondker Salim, María de Haro, Ismael Al‐Ramahi

This study shows that increasing KPNA levels reduces mutant Ataxin-1-induced neurodegeneration by retaining it in the cytoplasm and decreasing its nuclear accumulation.

bioRxiv · Jul 12, 2023 · PreprintNeuroscience

Modulating glial genes involved in synaptic function mitigates pathogenesis and behavioral deficits in a Drosophila model of Huntington's Disease

Tarik Seref Onur, Andrew Laitman, He Zhao, Ryan Keyho, Hye Min Kim, Jennifer P Wang, Megan L. Mair, Alma M. Perez, María de Haro, Huilan Wang, Ying‐Wooi Wan, Genevera I. Allen, Boxun Lu, Ismael Al‐Ramahi, Zhandong Liu, Juan Botas

The paper shows that reducing glial genes involved in synaptic function reduces disease pathology and behavioral issues in a Drosophila Huntington's Disease model.

bioRxiv · Nov 3, 2020 · PreprintSystems Biology