Author

Emilio Hirsch

University of TurinORCID 0000-0002-9073-6024Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Adi MoryiD· 1 shared Adolfo Gabriele MauroiD· 1 shared Alessandra GhigoiD· 1 shared André ReisiD· 1 shared Anlu CheniD· 1 shared Antje WieseneriD· 1 shared Antonio AbbateiD· 1 shared Antonio BerguaiD· 1 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperGenetics

Adi MoryORCID

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2 papers

Recent·Most cited

UNCONVENTIONAL INTERLEUKIN-1 SIGNALING IN CARDIAC DYSFUNCTION

Stefano A Toldo, Pratyush Narayan, Eleonora Mezzaroma, Alessandra Ghigo, Federico Damilano, Carlo Marchetti, Adolfo Gabriele Mauro, Emilio Hirsch, Benjamin W. Van Tassell, Antonio Abbate

This study investigates unconventional IL-1β signaling via PI3Kγ in cardiomyocytes, showing kinase-independent mechanisms link IL-1β to cardiac dysfunction and potential heart failure treatment insights.

bioRxiv · Mar 9, 2025 · PreprintImmunology

Mutations In PIK3C2A Cause Syndromic Short Stature, Skeletal Abnormalities, and Cataracts Associated With Ciliary Dysfunction

Dov Tiosano, Hagit N. Baris, Anlu Chen, Marrit M. Hitzert, Markus Schueler, Federico Gulluni, Antje Wiesener, Antonio Bergua, Adi Mory, Brett R. Copeland, Joseph G. Gleeson, Patrick Rump, Hester van Meer, Deborah A. Sival, Volker Haucke, , David A. Buchner

The paper identifies homozygous mutations in PIK3C2A causing a new Mendelian disorder with short stature, skeletal abnormalities, cataracts, and ciliary dysfunction.

PLoS Genetics · Apr 29, 2019Peer-reviewedGenetics