Author

Bin Huang

University of KentuckyORCID 0000-0002-9075-1531Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Bin Huang4 papers · China Pharmaceutical University

Biology Disciplines

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Physiology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Dan Hao· 2 shared Jianyao XueiD· 2 shared Ling GuoiD· 2 shared Misa ItoiD· 2 shared Qian WangiD· 2 shared Xiang‐An LiiD· 2 shared Ailing JiiD· 1 shared Albert Fliss· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersPhysiology

Dan Hao

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Author profile →

3 papers

Recent·Most cited

EZH1/2 inhibition improves immunotherapy response through MHC Class II de-repression and neutrophil reprogramming

Avery R. Childress, Dave-Preston I. Esoe, Xiulong Song, Christian M. Gosser, Yindan Lin, Daniel R. Plaugher, Tanner J. DuCote, Kassandra J. Naughton, Erika M. Skaggs, Harrison Yang, Ryan A. Goettl, Jinpeng Liu, Zhonglin Hao, Albert Fliss, Daisuke Honma, , Christine F. Brainson

This study shows that EZH1/2 inhibition with valemetostat enhances anti-PD1 immunotherapy in lung squamous cell carcinoma by increasing MHC Class II and reprogramming neutrophils.

bioRxiv · Aug 1, 2026 · PreprintCancer Biology

Identification of scavenger receptor BI as a scavenger of free heme that is essential for protection against hemolysis

Misa Ito, Jianyao Xue, Ling Guo, Dan Hao, Qian Wang, Alexander H. Williams, Chang‐Guo Zhan, Ailing Ji, Preetha Shridas, Wen Su, Shu Liu, Zhenheng Guo, M GONG, Scott M. Gordon, Bin Huang, , Xiang‐An Li

This study identifies scavenger receptor BI as a critical heme scavenger that protects against hemolysis, showing its therapeutic potential in hemolytic disorders.

Nature Communications · Jul 27, 2026Peer-reviewedPhysiology

Elevated plasma cholesterol causally improves sepsis outcome by promoting hepatic metabolic reprogramming

Qian Wang, Jianyao Xue, Ling Guo, Dan Hao, Misa Ito, Rianna Reese, Bin Huang, Congqing Wu, Xiang‐An Li

This study shows elevated plasma cholesterol improves sepsis survival by promoting hepatic metabolic reprogramming, as demonstrated in both human patients and mouse models.

Frontiers in Immunology · May 11, 2026Peer-reviewedPhysiology