Author

Johannes Andreas Hainfellner

Medical University of ViennaORCID 0000-0002-9148-958XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

Ábel VértesyiD· 1 shared Angela Maria Peer· 1 shared Ilaria MorassutiD· 1 shared Juergen A. KnoblichiD· 1 shared Julia S. F. ChuiD· 1 shared Maria NovatchkovaiD· 1 shared Martha FeuchtiD· 1 shared Mercedes F. ParedesiD· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Ábel VértesyORCID

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1 paper

Recent·Most cited

Cerebral organoid model reveals excessive proliferation of human caudal late interneuron progenitors in Tuberous Sclerosis Complex

Oliver L. Eichmüller, Nina S. Corsini, Ábel Vértesy, Ilaria Morassut, Theresa Scholl, Victoria‐Elisabeth Gruber, Angela Maria Peer, Julia S. F. Chu, Maria Novatchkova, Johannes Andreas Hainfellner, Mercedes F. Paredes, Martha Feucht, Juergen A. Knoblich

This study uses cerebral organoids to show that TSC2 mutations cause over-proliferation of caudal late interneuron progenitors, leading to Tuberous Sclerosis Complex.

Science · Jan 27, 2022Peer-reviewedNeuroscience