Author

Anabella Maria D. Galang

Columbia University Irving Medical CenterORCID 0000-0002-9245-0396Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:Arabidopsis· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

Aaron D. VinyiD· 1 shared Adrienne RoederiD· 1 shared Alexander Fisher· 1 shared Annamária KissiD· 1 shared Arezki BoudaoudiD· 1 shared Avilash Singh YadaviD· 1 shared Besmira Alija· 1 shared Claudia GebhardiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

Aaron D. VinyORCID

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2 papers

Recent·Most cited

Growth directions and stiffness across cell layers determine whether tissues stay smooth or buckle

Avilash Singh Yadav, Lilan Hong, Patrick M. Klees, Annamária Kiss, Manuel Petit, Xi He, Iselle M. Barrios, Michelle Heeney, Anabella Maria D. Galang, Richard S. Smith, Arezki Boudaoud, Adrienne Roeder

The paper shows that growth direction and stiffness differences between epidermal layers cause buckling in Arabidopsis sepals, and that restoring growth alignment and stiffness can prevent buckling.

Current Biology · Sep 1, 2026Peer-reviewedDevelopmental Biology

Altered chromatin accessibility mediated by somatic cohesin mutations amplifies FLI1-Menin output to promote leukemogenesis

Jane Jialu Xu, Viviana Scoca, Yi Chen, Yingqian Ada Zhan, Alexander Fisher, Eno-obong Udoh, Sebastian Fernando, Besmira Alija, John Pantazi, Varun S. Sudunagunta, Edna Stewart, Anabella Maria D. Galang, Michael Williams, Govind Bhagat, Claudia Gebhard, , Aaron D. Viny

This study shows that cohesin mutations alter chromatin accessibility, enhancing FLI1-Menin interaction and promoting leukemogenesis through therapeutic vulnerabilities.

bioRxiv · Apr 1, 2025 · PreprintCancer Biology