Author

Michael Williams

Cleveland Clinic

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

Aaron D. VinyiD· 1 shared Alexander Fisher· 1 shared Alexander G. StaffordiD· 1 shared Anabella Maria D. GalangiD· 1 shared Azeem ShardaiD· 1 shared Besmira Alija· 1 shared Christopher Johnson· 1 shared Claudia GebhardiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCancer Biology

Aaron D. VinyORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Kinetics of de novo Bone and Bone Marrow Niche Formation with Hybrid Click Cryogels

Sangmin Lee, Kwasi Adu‐Berchie, Azeem Sharda, Tania To, Nikolaos Dimitrakakis, Alexander G. Stafford, Katherine Sheehan, Christopher Johnson, Hamza Ijaz, Phoebe S. Kwon, Mark J. Cartwright, Sandy Elmehrath, Mary Catherine Skolfield, Nicholas C. Jeffreys, Des A. White, , David J. Mooney

This study investigates the kinetics of de novo bone and bone marrow niche formation using a hybrid click cryogel system, showing that BMP-2 dose, immune status, and sex influence development rates and hematopoietic cell populations.

Advanced Healthcare Materials · Feb 21, 2026Peer-reviewedBioengineering

Exploring the role of EMT in Ovarian Cancer Progression: Insights from a multiscale mathematical model

Samuel Oliver, Michael Williams, Mohit Kumar Jolly, Deyarina González, Gibin Powathil

This study uses a multiscale mathematical model to explore how EMT influences ovarian cancer progression in two cell lines, finding that EMT and cell cycle parameters significantly affect tumor dynamics.

npj Systems Biology and Applications · Apr 17, 2025Peer-reviewedCancer Biology

Altered chromatin accessibility mediated by somatic cohesin mutations amplifies FLI1-Menin output to promote leukemogenesis

Jane Jialu Xu, Viviana Scoca, Yi Chen, Yingqian Ada Zhan, Alexander Fisher, Eno-obong Udoh, Sebastian Fernando, Besmira Alija, John Pantazi, Varun S. Sudunagunta, Edna Stewart, Anabella Maria D. Galang, Michael Williams, Govind Bhagat, Claudia Gebhard, , Aaron D. Viny

This study shows that cohesin mutations alter chromatin accessibility, enhancing FLI1-Menin interaction and promoting leukemogenesis through therapeutic vulnerabilities.

bioRxiv · Apr 1, 2025 · PreprintCancer Biology