Author

Junping Yin

University Hospital BonnORCID 0000-0002-9270-2071Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Alaa OthmaniD· 1 shared Amina Abdelmageed· 1 shared Andreas ZiegleriD· 1 shared Andrew J. KuehiD· 1 shared Anke FähnrichiD· 1 shared Annika Voelkel· 1 shared Axel KünstneriD· 1 shared Beata GoldyniD· 1 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperGenetics

Alaa OthmanORCID

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2 papers

Recent·Most cited

Complement receptor-mediated uptake into renal mononuclear phagocytes promotes intracellular UPEC persistence and limits β-lactam efficacy in pyelonephritis

Beata Goldyn, Olena Babyak, Pranitha Rokkam, Yevheniia Minchuk, Marigona Sutaj, Clivia Lisowski, Nicolette Bluszcz, Daniela Klaus, Annika Voelkel, Amina Abdelmageed, Junping Yin, Andrew J. Kueh, Huiyun Hu, Isabelle Mueller, Marco J. Herold, , Christian Kurts

This study identifies renal mononuclear phagocytes as an intracellular reservoir for UPEC in pyelonephritis, showing that complement receptor-mediated entry promotes bacterial persistence and limits β-lactam efficacy.

bioRxiv · Jul 9, 2026 · PreprintMicrobiology

Low-level mitochondrial heteroplasmy modulates DNA replication, glucose metabolism and lifespan in mice

Misa Hirose, Paul Schilf, Yask Gupta, Kim Zarse, Axel Künstner, Anke Fähnrich, Hauke S. Busch, Junping Yin, Marvin N. Wright, Andreas Ziegler, Marie Vallier, Meriem Belheouane, John F. Baines, Diethard Tautz, Kornelia Johann, , Saleh Mohamed Ibrahim

This study shows that high mitochondrial heteroplasmy in mice reduces lifespan and impairs glucose metabolism.

Scientific Reports · Apr 11, 2018Peer-reviewedGenetics