Author

François Korbmacher

European Molecular Biology LaboratoryORCID 0000-0002-9977-7885Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

Hannah FleckensteiniD· 3 shared Maria BernabeuiD· 3 shared Rory K. M. LongiD· 3 shared Alina BatzillaiD· 2 shared Borja López-GutiérreziD· 2 shared Livia PiattiiD· 2 shared Yannick SchwabiD· 2 shared Daniel SchraivogeliD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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3 shared papersMicrobiology

Hannah FleckensteinORCID

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3 papers

Recent·Most cited

ETS-guided iPSC-endothelial models recapitulate malaria pathogenesis

François Korbmacher, Rory K. M. Long, Hannah Fleckenstein, Patryk Poliński, Dennis Crusius, Livia Piatti, Borja López-Gutiérrez, Alina Batzilla, Vikas Trivedi, Miki Ebisuya, Maria Bernabeu

This study develops an iPSC-based model with enhanced endothelial identity to study malaria parasite interactions and pathogenesis in vitro.

EMBO Molecular Medicine · Jun 13, 2026Peer-reviewedMicrobiology

Plasmodium falciparum impairs Ang-1 secretion by pericytes in a 3D brain microvessel model

Rory K. M. Long, François Korbmacher, Paolo Ronchi, Hannah Fleckenstein, Martin Schorb, Waleed Mirza, Mireia Mallorquí, Ruth Aguilar, Gemma Moncunill, Yannick Schwab, Maria Bernabeu

This paper shows that Plasmodium falciparum impairs Ang-1 secretion by pericytes in a 3D brain microvessel model, leading to vascular dysfunction that can be partially reversed with Ang-1 or AKB-9778.

EMBO Molecular Medicine · Oct 16, 2025Peer-reviewedMicrobiology

Plasmodium falciparum egress disrupts endothelial junctions and activates JAK-STAT signaling in a microvascular 3D blood-brain barrier model

Livia Piatti, Alina Batzilla, Fumio Nakaki, Hannah Fleckenstein, François Korbmacher, Rory K. M. Long, Daniel Schraivogel, John A. Hawkins, Tais Romero-Uruñuela, Borja López-Gutiérrez, Sílvia Sanz, Yannick Schwab, Lars Michael Steinmetz, James A. Sharpe, Maria Bernabeu

This study shows that Plasmodium falciparum egress products disrupt BBB endothelial junctions and activate JAK-STAT signaling, leading to increased permeability and suggesting therapeutic targets.

Nature Communications · Aug 6, 2025Peer-reviewedMicrobiology