Author

Josef Vágner

University of ArizonaORCID 0000-0003-1289-0234Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Assay:proteomics· 1

Frequent Co-Authors

Ayesha AhmadiD· 3 shared Gregory O. DussoriD· 3 shared Moeno KumeiD· 3 shared Scott A. Boitano· 3 shared Theodore John PriceiD· 3 shared Juliet M. MwirigiiD· 2 shared Pradipta R. RayiD· 2 shared Shayne N. HassleriD· 2 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 44 mutual co-author links

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3 shared papersNeuroscience

Ayesha AhmadORCID

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3 papers

Recent·Most cited

NeuroscienceJournal of PainPeer-reviewedJun 12, 2023

Protease-Activated Receptor 2 (PAR2) expressed in sensory neurons contributes to signs of pain and neuropathy in paclitaxel treated mice

Moeno Kume, Ayesha Ahmad, Kathryn DeFea…·Theodore John Price

This study shows that PAR2 in sensory neurons contributes to paclitaxel-induced pain and neuropathy in mice, and that targeting PAR2 can alleviate these symptoms.

15 citations

mouseexperimental assay

𝕏 PostBluesky

NeuroscienceJournal of PainPeer-reviewedJan 12, 2021

A role for protease activated receptor type 3 (PAR3) in nociception demonstrated through development of a novel peptide agonist

Juliet M. Mwirigi, Moeno Kume, Shayne N. Hassler…·Theodore John Price

The paper develops a PAR3 agonist and shows it induces mechanical hypersensitivity in mice, suggesting PAR3's role in nociception and plasticity.

11 citations

mouseproteomics

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NeuroscienceJCI InsightPeer-reviewedApr 30, 2020

The cellular basis of protease activated receptor type 2 (PAR2) evoked mechanical and affective pain

Shayne N. Hassler, Moeno Kume, Juliet M. Mwirigi…·Theodore John Price

This study identifies a specific subset of sensory neurons expressing PAR2 that mediate mechanical and affective pain behaviors, clarifying the cellular basis of PAR2-evoked pain.

25 citations

mousebulk RNA-seq

𝕏 PostBluesky