Author

Parisa Shabani

University of MichiganORCID 0000-0003-2345-9052Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:metabolism & bioenergetics· 1 Assay:proteomics· 1

Frequent Co-Authors

Afnan AlzamrooniiD· 3 shared Ahmed Abdel‐LatifiD· 3 shared Tahra Kaur Luther· 3 shared Anand Prakash SinghiD· 2 shared Rachel Lopez‐Schenk· 2 shared Rajesh Kumar ChaudharyiD· 2 shared Anand P. Singh· 1 shared Andrew Robbins· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 34 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersImmunology

Afnan AlzamrooniORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

ImmunologybioRxivPreprintAug 30, 2026

Autotaxin Inhibition Ameliorates HFpEF Phenotype By Reducing LPA-Mediated Systemic Inflammation And Cardiac Remodeling

Rajesh Chaudhary, Andrew Robbins, Anand P. Singh…·Ahmed Abdel‐Latif

This study shows autotaxin inhibition reduces LPA-mediated inflammation and cardiac remodeling in HFpEF, improving diastolic function and fibrosis in mice and showing altered lysophospholipid profiles in humans.

DOI 10.64898/2026.08.26.747366

mouseproteomics

𝕏 PostBluesky

ImmunologybioRxivPreprintJun 9, 2026

Soluble Urokinase Plasminogen Activator Receptor Primes Macrophages and Worsens Heart Failure with Preserved Ejection Fraction

Anand Prakash Singh, Parisa Shabani, Anis Ismail…·Ahmed Abdel‐Latif

This paper shows that elevated suPAR worsens HFpEF by priming macrophages and amplifying inflammatory responses, identifying a therapeutic target for anti-suPAR antibodies.

DOI 10.64898/2026.06.04.730230

mousebulk RNA-seq

𝕏 PostBluesky

Cell BiologybioRxivPreprintNov 27, 2025

Targeting Metabolic Dysfunction and Inflammation with Sotagliflozin Reverses Diastolic Dysfunction in Experimental HFpEF

Parisa Shabani, Tahra Kaur Luther, Rajesh Kumar Chaudhary…·Ahmed Abdel‐Latif

This study shows that sotagliflozin improves diastolic dysfunction in HFpEF by enhancing lipid metabolism and reducing inflammation.

DOI 10.1101/2025.11.27.691021

mousebulk RNA-seq

𝕏 PostBluesky