Daniel J. Liebl

This study identifies a sex-specific reactivation of developmental pruning processes as a cause of cognitive dysfunction after brain trauma and shows the pathway is reversible.

This paper introduces two marker-less kinematic analysis systems for assessing mouse locomotor function after spinal cord injury, validated against traditional methods and showing high sensitivity and accuracy.

This paper identifies FEM-1689 as a σ2R/TMEM97 ligand that alleviates neuropathic pain by inhibiting the integrated stress response in mice and human sensory neurons.

This study shows Eph:ephrin signaling maintains progenitor identities in the ventral spinal cord by regulating transcription factor expression.