Author

Prosanta Singha

University of Eastern FinlandORCID 0000-0003-2773-4330Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Minna Unelma KaikkoneniD· 2 shared Tiit ÖrdiD· 2 shared Adriana BlazeskiiD· 1 shared Anu ToropaineniD· 1 shared Åsa SegerstolpeiD· 1 shared Calum A. MacRaeiD· 1 shared Carles A. BoixiD· 1 shared Clint L. MilleriD· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersMolecular Biology

Minna Unelma KaikkonenORCID

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2 papers

Recent·Most cited

Molecular BiologyCell Reports MedicinePeer-reviewedMay 1, 2024

Cardiovascular disease causes proinflammatory microvascular changes in the human right atrium

Suvi Linna-Kuosmanen, Eloi Schmauch, Kyriaki Galani…·M Kellis

The paper uses single-nuclei RNA-seq and spatial transcriptomics to analyze gene expression changes in the right atrium of patients with ischemic heart disease, revealing inflammatory microvascular dysfunction and altered tissue composition.

27 citations

humanscRNA-seq

𝕏 PostBluesky

GeneticsbioRxivPreprintJan 24, 2022

Genetic regulation of human aortic smooth muscle cell gene expression and splicing predict causal coronary artery disease genes

Rédouane Aherrahrou, Dillon Lue, R. Noah Perry…·Mete Civelek

The paper identifies eQTLs and sQTLs in human aortic SMCs that colocalize with CAD GWAS loci, highlighting splicing regulation as a mechanism for CAD risk.

4 citations

humanbulk RNA-seq

𝕏 PostBluesky