Author

Robert D. Cardiff

UC Davis Comprehensive Cancer CenterORCID 0000-0003-3088-8816Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Alexander D. BorowskyiD· 2 shared Joshua C. SnyderiD· 2 shared Alejandra V. PatinoiD· 1 shared Bruce W. RogersiD· 1 shared Edwin J. Allen· 1 shared Henry Chapman· 1 shared Herbert Kim LyerlyiD· 1 shared Hidetoshi MoriiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersCancer Biology

Alexander D. BorowskyORCID

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2 papers

Recent·Most cited

Cancer BiologyDisease Models & MechanismsPeer-reviewedMar 1, 2025

Nonlinear progression during the occult transition establishes cancer lethality

Joshua D. Ginzel, Henry Chapman, Joelle E. Sills…·Joshua C. Snyder

This study shows that cancer progression is nonlinear, with a critical occult transition leading to lethal tumors before detection, challenging the linear model of cancer growth.

8 citations

mousestatistical model

𝕏 PostBluesky

Cancer BiologybioRxivPreprintFeb 2, 2025

Adaptive selection of p53 mutation metaplastic phenotypes in estrogen-independent progression of ER+ tumors: A mechanism for acquired resistance to hormonal therapy.

Hyeongsun Moon, Hidetoshi Mori, Jane Chen…·Alexander D. Borowsky

This study shows that ER+ breast cancers can develop resistance to hormonal therapy through clonal selection of metaplastic p53-mutated basal cells in Stat1-/- mice.

DOI 10.1101/2025.02.02.636128

mousebulk RNA-seq

𝕏 PostBluesky