Paul E. Turner

The paper investigates phage susceptibility variation in chronic Pseudomonas aeruginosa lung infections and finds it varies within patients, possibly due to cell-surface features, challenging phage therapy efficacy.

The paper shows that diverse microbial communities promote phage-bacteria coexistence by limiting resistance evolution in bacteria, increasing phage persistence.

This study shows prophage infection influences bacterial thermal ecology and evolution by altering thermal adaptation and population dynamics across temperatures.

This study shows that filamentous phage mutations drive eco-evolutionary feedbacks in Pseudomonas aeruginosa by promoting phage resistance and altering key traits.

This study examines how host changes affect viral evolution by analyzing allele frequency dynamics and finding that Taylor's power law governs mutation distribution during host replacement.

This study shows that phage resistance in E. coli leads to diverse single-cell motility outcomes and signaling pathway changes, revealing trade-offs and trade-ups in motility and chemotaxis.

This paper introduces a high-throughput method to quantify phage attachment to bacterial cells using fluorescence microscopy and particle tracking, revealing single-virus dynamics and enabling rapid screening.

The paper identifies eight Enterobacter jumbo phages with broad host ranges across pathogenic Gammaproteobacteria, suggesting potential as antibiotic alternatives.

The paper identifies bacterial receptor genes determining phage host range, showing defense mechanisms are not predictive of susceptibility.

This study investigates how lytic bacteriophages interact with respiratory epithelial cells and their effects on cytokine secretion, highlighting the need to consider mammalian responses in phage therapy.