Author

Gregory F. Wu

Washington University in St. LouisORCID 0000-0003-3588-0637Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Immunology· 2 Neuroscience· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Assay:flow cytometry· 2 Research Focus:host-pathogen & immune response· 2 Approach:wet-lab / biochemical assay· 2 Assay:CITE-seq· 1

Frequent Co-Authors

Abraham Ogbaslase· 1 shared Alexandria J. SturtziD· 1 shared Angela S. Archambault· 1 shared Benedict E Ridore· 1 shared Brian KimiD· 1 shared Hongzhen HuiD· 1 shared Huan Zhou· 1 shared Jennifer L. GommermaniD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

Abraham Ogbaslase

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

ImmunologybioRxivPreprintApr 20, 2026

Distinct contribution of autoreactive B cell Bruton's tyrosine kinase signaling to neuroinflammation

Abraham Ogbaslase, Angela S. Archambault, Kia M. Barclay…·Gregory F. Wu

This study shows that deleting BTK in B cells reduces EAE severity by impairing autoreactive B cell functions, supporting BTK as a therapeutic target in MS.

DOI 10.64898/2026.04.14.718534

mouseCITE-seq

𝕏 PostBluesky

ImmunologybioRxivPreprintJan 27, 2025

Unconventionally primed type 1 follicular helper T cells are required for long-lived IgA plasma cell development following mucosal viral infection

Kei Haniuda, Natalie M. Edner, Yuko Makita…·Jennifer L. Gommerman

This study identifies unconventionally primed TFH1 cells as essential for generating IgA+ long-lived plasma cells during mucosal viral infections.

DOI 10.1101/2025.01.27.635134

mouseflow cytometry

𝕏 PostBluesky

NeuroscienceCell ReportsPeer-reviewedMar 22, 2023

MrgprA3-expressing pruriceptors drive pruritogen-induced alloknesis through mechanosensitive Piezo2 channel

Ping Lü, Yonghui Zhao, Zili Xie…·Hongzhen Hu

The paper identifies TRPV1+/MrgprA3+ prurioceptors and Piezo2 channel signaling as drivers of pruritogen-induced alloknesis.

28 citationstop 3%

mouseexperimental assay

𝕏 PostBluesky