Stefan Schulte‐Merker

This study identifies SVEP1 as a mediator of ANG2-TIE1 binding, leading to TIE1 phosphorylation and downstream signaling in lymphatic endothelial cells.

The paper integrates ATAC-seq and RNA-seq data from zebrafish osteoblasts to identify chromatin and transcriptomic signatures and mechanisms of entpd5a regulation.

This study shows that Shp2 is essential for lymphatic endothelial cell differentiation in zebrafish embryos, without affecting venous angiogenesis.

The paper identifies Angpt1/Tie1 signaling as essential for trunk lymphatic vessel development in zebrafish, showing Tie1 regulates Foxo1 localization and modulates Vegfc/Vegfr3 signaling.

This study shows that neural plate progenitors contribute to both anterior and posterior pituitary cells in zebrafish and mouse, challenging the traditional view of pituitary cell origins.

This study identifies a genetic interaction between svep1 and tie1 in zebrafish lymphatic development, independent of Vegfc, and links it to human glaucoma.

This study identifies that arterial mural cells provide survival and guidance cues via Cxcl12a/b and Vegfc to promote lymphatic endothelial cell migration in zebrafish.

This study identifies Svep1 as a factor that stabilizes vascular anastomosis in zebrafish embryos under reduced flow conditions.

The paper compares brain lymphatic endothelial cells (BLECs) to scavenging endothelial cells (SECs) in the zebrafish and identifies Mrc1a as a key receptor for cargo uptake in BLECs.

The paper investigates how PI3K signaling affects survival and stemness of zebrafish HSPCs during EHT, finding that both loss of Pten and PI3K inhibition induce apoptosis, while surviving cells show reduced stemness.

The paper describes the discovery of transient NO-containing spherical structures in zebrafish cerebral endothelial cells during development, showing their dependence on Notch signaling.

The paper identifies Endothelin receptor Aa as a regulator of APSC quiescence in zebrafish, linking blood vessel signals to pigment cell differentiation.

The paper identifies a wound-specific notochord sheath cell subpopulation expressing Wilms Tumor 1b that forms a stopper-like structure during vertebra repair in zebrafish.