Kyla S. Ost

This study shows that Candida albicans supports Group B Streptococcus vaginal persistence and pathogenesis through physical and metabolic interactions.

This study identifies Awp11, an adhesin-like effector in Candida glabrata, as a key determinant of strain-dependent immunogenicity and gut colonization.

This study shows that bacterial metabolites alter C. albicans cell wall composition, affecting antifungal resistance and immune recognition.

The paper identifies a non-genetic, heritable mechanism of drug resistance in Candida albicans triggered by subtherapeutic fluconazole exposure, which can be reversed and involves prion-like epigenetic processes.