Author

Anne Quatraccioni

University of BonnORCID 0009-0007-0061-8571Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Albert J. BeckeriD· 3 shared Julika PitschiD· 3 shared Silvia Cases‐CunilleraiD· 3 shared Susanne SchochiD· 3 shared Dirk DietrichiD· 2 shared Karen M. J. van LooiD· 2 shared Barbara K. RobensiD· 1 shared Heinz BeckiD· 1 shared

Activity by Year

2021-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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3 shared papersNeuroscience

Albert J. BeckerORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

NeurosciencebioRxivPreprintAug 18, 2026

Single-cell profiling reveals tumor grade-dependent immune remodeling in BRAFV600E-driven glioneuronal tumors

Kittitach Sri-ngern-ngam, Philipp Müller, Anne Quatraccioni…·Silvia Cases‐Cunillera

This study uses single-cell transcriptomics to show that BRAFV600E-driven glioneuronal tumors have distinct immune profiles based on tumor grade, with SPP1 playing a key role in high-grade tumors.

DOI 10.64898/2026.08.10.743180

mousescRNA-seq

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NeuroscienceJournal of NeurosciencePeer-reviewedAug 16, 2021

Ste20-like kinase is critical for inhibitory synapse maintenance and its deficiency confers a developmental dendritopathy

Susanne Schoch, Anne Quatraccioni, Barbara K. Robens…·Albert J. Becker

This study identifies SLK as critical for dendritic complexity and inhibitory synapse maintenance in neurons, with implications for human cortical malformations.

9 citations

mousehuman

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NeurosciencebioRxivPreprintFeb 22, 2021

Heterogeneity and excitability of BRAFV600E-induced tumors is determined by PI3K/mTOR-signaling state and Trp53-loss

Silvia Cases‐Cunillera, Karen M. J. van Loo, Julika Pitsch…·Albert J. Becker

This study identifies how PI3K/mTOR signaling and Trp53 loss determine the heterogeneity and excitability of BRAFV600E-induced brain tumors in mice.

DOI 10.1101/2021.02.22.432030

mouseexperimental assay

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