Author

Ivan Gallotta

Institute of Genetics and BiophysicsORCID 0009-0008-1521-2029Unclaimed ORCID profile · follow to get notified of new preprints

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Genetics· 2

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Organism:C. elegans· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:cell-cell communication· 1 Organism:Drosophila· 1 Research Focus:gene regulatory network inference· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

Elia Di SchiaviiD· 2 shared Pamela SantonicolaiD· 2 shared Alessandra Galati· 1 shared Alessandro EspositoiD· 1 shared Alessio ColantoniiD· 1 shared Brunhilde WirthiD· 1 shared Caitlin M. RoakeiD· 1 shared Cristiano De PittàiD· 1 shared

Activity by Year

2020-2026 · peak 1/yr

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10 top collaborators · 33 mutual co-author links

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2 shared papersGenetics

Elia Di SchiaviORCID

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2 papers

Recent·Most cited

GeneticsLife Science AlliancePeer-reviewedJul 13, 2026

A Spinal Muscular Atrophy Model Reveals Both Developmental and Degenerative Neuronal Defects in C. elegans

Sara Savaheli, Ivan Gallotta, Pamela Santonicola…·Denis Dupuy

This study uses a C. elegans model to show that SMN-1 deficiency causes developmental and degenerative defects in motor and touch receptor neurons, suggesting conserved SMA pathology.

DOI 10.26508/lsa.202603647

C. elegansexperimental assay

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GeneticsbioRxivPreprintOct 27, 2020

TGS1 controls snRNA 3' end processing, prevents neurodegeneration and ameliorates SMN-dependent neurological phenotypes in vivo

Lü Chen, Caitlin M. Roake, Paolo Maccallini…·Grazia Daniela Raffa

This study shows TGS1 is essential for snRNA 3' end processing and its loss causes neurodegeneration, which can be mitigated by human TGS1 expression in model organisms.

2 citations

C. elegansDrosophila

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