Author

Nela Fialova

British Heart FoundationORCID 0009-0009-0497-3882Unclaimed ORCID profile · follow to get notified of new preprints

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:spatial transcriptomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Maurits A. JanseniD· 2 shared Ross J. Lennen· 2 shared Alessandra GranataiD· 1 shared Alexandra V. PokhilkoiD· 1 shared Anna WilliamsiD· 1 shared Audrey ChagnotiD· 1 shared Axel MontagneiD· 1 shared Conor McQuaidiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersNeuroscience

Maurits A. JansenORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintMay 13, 2026

Pericyte Loss Reprogrammes Capillary Endothelium and Drives White Matter Injury in Small Vessel Disease

Dorota Stefancova, Audrey Chagnot, Michael D.E. Sewell…·Axel Montagne

1 citation

humanbulk RNA-seq

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NeurosciencebioRxivPreprintApr 4, 2025

Cerebral small vessel disease due to Col4a1 mutations include cognitive impairment and white matter defects that can be modulated by targeting protein folding.

Gaia Brezzo, Alexandra V. Pokhilko, Jon Moss…·Karen J. Horsburgh

This study identifies cognitive impairment and white matter defects in a mouse model of cerebral small vessel disease caused by Col4a1 mutations and shows that targeting protein folding can alleviate these issues.

DOI 10.1101/2025.04.04.647207

mouseexperimental assay

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