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Cell Death and DiseaseCancer BiologyPeer-reviewed journal article

Atypical contribution of caspase-3 to melanoma cancer cell motility by regulation of coronin 1B activity

Kévin BerthenetiD, Serena DiazziiD, Catherine JamardiD, Kinga B. StopaiD, Clara GiliD, Stefan DraganiD, Deborah FanfoneiD, Trang Thi Minh NguyeniD, Nathalie Al, François VirardiD, Olivier MeuretteiD, Nikolay PopgeorgieviD, Héctor Hernández‐VargasiD, Julien AblainiD, Gabriel IchimiD

PDFDOIPreprint

Abstract

Recent studies have unveiled unexpected connections between cell death and cell motility. While traditionally recognized for their pro-apoptotic roles, caspases have emerged as regulators of physiological processes beyond cell death, including cellular differentiation and motility. In some particularly aggressive cancers like melanoma, caspase-3, a prominent executioner caspase, is unexpectedly and inexplicably highly expressed. Here, we describe a novel non-apoptotic role for caspase-3 in melanoma cell motility. Through comprehensive molecular and cellular analyses, we demonstrate that caspase-3 is constitutively associated with the cytoskeleton and crucially regulates melanoma cell migration and invasion in vitro and in vivo. Mechanistically, caspase-3 interacts with and modulates the activity of coronin 1B, a key regulator of actin polymerization, thereby promoting melanoma cell motility, independently of its apoptotic protease function. Furthermore, we identify specificity protein 1 (SP1) as a transcriptional regulator of CASP3 expression, and show that its inhibition reduces caspase-3 expression and impairs melanoma cell migration. Overall, this study provides insights into the multifaceted roles of caspase-3 in cancer progression, highlighting its relevance as a novel target for anti-metastatic therapies.

Citations
2
FWCI
0.48
Field percentile
top 43%

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