A. Di Pietro

Cytosolic pH controls fungal MAPK signaling and pathogenicity

This study shows that cytosolic pH controls MAPK signaling and pathogenicity in fungi by modulating phosphorylation levels and sphingolipid metabolism.

Components of TOR and MAP kinase signaling control chemotropism and pathogenicity in the fungal pathogen Verticillium dahliae

This study identifies key regulators of chemotropism in Verticillium dahliae, showing their roles in nutrient sensing, plant signal detection, and pathogenicity.

Impairment of the cellulose degradation machinery enhances fungal virulence but limits reproductive fitness

The paper shows that impairing cellulose degradation in Fusarium oxysporum increases virulence but reduces reproductive fitness.

Conserved secreted effectors determine endophytic growth and multi-host plant compatibility in a vascular wilt fungus

This study identifies conserved fungal effectors that enable multi-host plant colonization and compatibility in the vascular wilt fungus Fusarium oxysporum.

Integration of chemosensing and carbon catabolite repression impacts fungal enzyme regulation and plant associations

This study investigates how Trichoderma reesei senses glucose and regulates enzyme production and development in response to plant environments.

Marchantia polymorpha model reveals conserved infection mechanisms in the vascular wilt fungal pathogen Fusarium oxysporum

This paper shows that the vascular wilt fungus Fusarium oxysporum can infect the non-vascular plant Marchantia polymorpha, revealing conserved pathogenic mechanisms and dispensable lineage-specific effectors.

A homologue of the mammalian tumour suppressor protein PTEN is a functional lipid phosphatase and required for chemotaxis in filamentous fungi

This study identifies TepA, a fungal homologue of PTEN, as a functional lipid phosphatase required for chemotaxis in filamentous fungi.

The histone methyltransferase DOT1L is essential for humoral immune responses

This study shows that DOT1L is essential for B cell development and humoral immune responses, as its deletion leads to impaired germinal center formation and reduced antibody production.