Author

A. J. Rogers

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:scRNA-seq· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Approach:deep learning· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. J. Wilk· 2 shared C. A. Blish· 2 shared N. Q. Zhao· 2 shared A. C. Lewis· 1 shared A. E. Tron· 1 shared A. L. Blomkalns· 1 shared A. Rustagi· 1 shared A. S. Trigos· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersImmunology

A. J. Wilk

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Inhibition of mutant IDH1 promotes cycling of acute myeloid leukemia stem cells

E. Gruber, J. So, A. C. Lewis, R. Franich, R. Cole, L. G. Martelotto, A. J. Rogers, E. Vidacs, P. Fraser, K. Stanley, L. Jones, A. S. Trigos, N. Thio, J. Li, B. Nicolay, , L. M. Kats

This study shows that inhibiting mutant IDH1 in AML promotes leukemic stem cell cycling and sensitizes the disease to azacitidine, suggesting a combination therapy approach.

bioRxiv · Apr 6, 2022Cancer Biology

Multi-omic profiling reveals widespread dysregulation of innate immunity and hematopoiesis in COVID-19

A. J. Wilk, M. J. Lee, B. Wei, B. Parks, R. Pi, G. J. Martinez-Colon, T. Ranganath, N. Q. Zhao, S. Taylor, W. Becker, Stanford COVID-19 Biobank, D. Jimenez-Morales, A. L. Blomkalns, R. O'Hara, E. A. Ashley, , C. A. Blish

Single-cell multi-omic profiling identifies immune dysregulation in severe COVID-19, including neutrophil hyperactivation and anti-inflammatory monocytes.

bioRxiv · Dec 18, 2020Immunology

Additional analyses exploring the hypothesized transdifferentiation of plasmablasts to developing neutrophils in severe COVID-19

A. J. Wilk, A. Rustagi, N. Q. Zhao, B. A. Martin, A. J. Rogers, C. A. Blish

The paper responds to criticisms of a previous study on transdifferentiation of plasmablasts to neutrophils in severe COVID-19 using single-cell transcriptomic data.

bioRxiv · Oct 15, 2020 · PreprintImmunology