Author

A. J. Ronk

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Bukreyev· 3 shared A. Antanasijevic· 1 shared A. D. Jayaprakash· 1 shared A. N. Prasad· 1 shared A. Ward· 1 shared C. F. Basler· 1 shared C. Mire· 1 shared C. Wirblich· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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3 shared papersImmunology

A. Bukreyev

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Author profile →

3 papers

Recent·Most cited

Lassa virus glycoprotein nanoparticles elicit a neutralizing antibody that defines a new site of vulnerability

P. J. M. Brouwer, A. Antanasijevic, A. J. Ronk, H. Mulller-Krauter, Y. Watanabe, M. Claireaux, N. M. Lloyd, T. P. L. Bijl, H. R. Perrett, T. H. Steijaert, J. A. Burger, M. M. van Haaren, K. Sliepen, M. van Gils, M. Crispin, , R. W. Sanders

This study develops Lassa virus glycoprotein nanoparticles that stabilize GPC trimers and elicit neutralizing antibodies targeting a new vulnerability site.

bioRxiv · Mar 28, 2022 · PreprintImmunology

A single dose of replication-competent VSV-vectored vaccine expressing SARS-CoV-2 S1 protects against virus replication in a hamster model of severe COVID-19

D. C. Malherbe, D. Kurup, C. Wirblich, A. J. Ronk, C. Mire, N. Kuzmina, N. Shaik, S. Periasamy, M. A. Hyde, J. M. Williams, P.-Y. Shi, M. J. Schnell, A. Bukreyev

A single dose of the VSV-vectored vaccine ConVac expressing SARS-CoV-2 S1 subunit protects hamsters from weight loss and viral replication after SARS-CoV-2 challenge.

bioRxiv · Jan 29, 2021Immunology

Marburg and Ebola virus infections elicit a complex, muted inflammatory state in bats

A. D. Jayaprakash, A. J. Ronk, A. N. Prasad, M. F. Covington, K. R. Stein, T. M. Schwarz, S. Hekmaty, K. A. Fenton, T. W. Geisbert, C. F. Basler, A. Bukreyev, R. Sachidanandam

This study investigates how Marburg and Ebola viruses elicit a complex, muted inflammatory response in bats, revealing mechanisms that may explain their non-pathogenicity in these animals.

bioRxiv · Apr 13, 2020Microbiology