Author

A. Kahana

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Evolutionary Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:mouse· 1

Frequent Co-Authors

A. Comba· 1 shared A. E. Argento· 1 shared C. Abel· 1 shared C. E. Brown· 1 shared C. G. Kleer· 1 shared D. B. Zamler· 1 shared D. Despotovic· 1 shared D. S. Tawfik· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. Comba

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Identifying the Link Between Chemical Exposures and Breast Cancer in African American Women via ToxCast High Throughput Screening Data

K. Polemi, V. K. Nguyen, J. Heidt, A. Kahana, O. Jolliet, J. Colacino

This study identifies chemicals with breast cancer-related biological activity that are disproportionately exposed in African American women using ToxCast data and biomonitoring.

bioRxiv · Jan 22, 2021Pharmacology & Toxicology

Spatiotemporal analysis of glioma heterogeneity reveals Col1A1 as an actionable 1 target to disrupt tumor mesenchymal differentiation, invasion and malignancy

A. Comba, S. M. Faisal, P. J. Dunn, A. E. Argento, T. C. Hollon, W. Al-Holou, M. L. Varela, D. B. Zamler, G. L. Quass, P. F. Apostolides, C. Abel, C. E. Brown, P. E. Kish, A. Kahana, C. G. Kleer, , P. R. Lowenstein

The paper identifies COL1A1 as a key driver of glioma mesenchymal differentiation and invasion, showing its inhibition disrupts tumor structure and improves survival.

bioRxiv · Dec 1, 2020 · PreprintCancer Biology

Polyamines Mediate Folding of Primordial Hyper-Acidic Helical Proteins

D. Despotovic, L. M. Longo, E. Aharon, A. Kahana, T. Scherf, I. Gruic-Sovulj, D. S. Tawfik

This study shows polyamines induce folding of a hyper-acidic ancestral protein, suggesting they may have functioned as chemical chaperones in early life.

bioRxiv · Oct 1, 2020Evolutionary Biology