Author

A. M. Obeidat

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1 Assay:imaging· 1

Frequent Co-Authors

A.-M. Malfait· 3 shared J. Li· 2 shared R. E. Miller· 2 shared S. Ishihara· 2 shared B. Hu· 1 shared C. B. Little· 1 shared C. Rasner· 1 shared C. Scanzello· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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3 shared papersPhysiology

A.-M. Malfait

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Author profile →

3 papers

Recent·Most cited

Recommendations For a Standardized Approach to Histopathologic Evaluation of Synovial Membrane in Murine Models of Experimental Osteoarthritis

A. M. Obeidat, S. Y. Kim, K. G. Burt, B. Hu, J. Li, S. Ishihara, R. Xiao, R. E. Miller, C. B. Little, A.-M. Malfait, C. Scanzello

The paper develops a standardized histopathologic evaluation approach for synovial membrane in murine OA models, showing good inter-reader agreement and consistent findings across stains.

bioRxiv · Oct 14, 2023 · PreprintPathology

Sexual dimorphism of the synovial transcriptome underpins greater PTOA disease severity in male mice following joint injury

R. F. Bergman, L. Lammlin, L. Junginger, E. Farrell, S. Goldman, R. Darcy, C. Rasner, A. M. Obeidat, A.-M. Malfait, R. Miller, T. Maerz

This study shows male mice have worse joint damage and persistent synovial inflammation after ACLR injury compared to females.

bioRxiv · Nov 30, 2022Physiology

Piezo2 expressing nociceptors mediate mechanical sensitization in experimental osteoarthritis

A. M. Obeidat, M. J. Wood, S. Ishihara, J. Li, L. Wang, D. Ren, D. A. Bennett, R. J. Miller, A.-M. Malfait, R. E. Miller

This study shows that Piezo2 is essential for mechanical pain sensitization in osteoarthritis, as Piezo2-deficient mice are protected from mechanical hypersensitivity in two models.

bioRxiv · Mar 12, 2022Physiology