Author

A. Martinez-Ruiz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:metabolomics· 2 Assay:proteomics· 2 Approach:biophysical modeling· 1 Research Focus:cell signaling & trafficking· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

A. Cogolludo· 2 shared C. Choya-Foces· 2 shared A. Bogdanova· 1 shared A. Cortes· 1 shared A. Izquierdo-Alvarez· 1 shared A. Martinez-del-Pozo· 1 shared A. Palomino-Antolin· 1 shared A. V. Lechuga-Vieco· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

A. Cogolludo

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cigarette smoke induces pulmonary arterial dysfunction through an imbalance in the guanylyl cyclase redox status.

J. Sevilla-Montero, J. Pino-Fadon, O. Munar-Rubert, M. Villegas-Esguevillas, B. Climent, M. Agro, C. Choya-Foces, A. Martinez-Ruiz, E. Balsa, C. Munoz, R. M. Gomez-Punter, E. Vazquez-Espinosa, A. Cogolludo, M. J. Calzada

This study shows cigarette smoke extract impairs pulmonary artery function by disrupting mitochondrial redox balance and guanylyl cyclase activity, suggesting mitochondrial-targeted antioxidants could treat COPD-related pulmonary hypertension.

bioRxiv · Feb 21, 2022 · PreprintCell Biology

Mitochondrial Na+ controls oxidative phosphorylation and hypoxic redox signalling

P. Hernansanz-Agustin, C. Choya-Foces, S. Carregal-Romero, E. Ramos, T. Oliva, T. Villa-Pina, L. Moreno, A. Izquierdo-Alvarez, J. D. Cabrera-Garcia, A. Cortes, A. V. Lechuga-Vieco, P. Jadiya, E. Navarro, E. Parada, A. Palomino-Antolin, , A. Martinez-Ruiz

This study shows that mitochondrial Na+ acts as a second messenger regulating OXPHOS and ROS production through interactions with phospholipids during hypoxia.

bioRxiv · Oct 31, 2019Biochemistry