Author

A. Pisconti

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

F. K. Jones· 2 shared A. Butcher· 1 shared A. Daroszewska· 1 shared A. G. Kay· 1 shared A. Phillips· 1 shared A. Prior· 1 shared A. R. Jones· 1 shared A. Stefan· 1 shared

Activity by Year

2019-2020 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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2 shared papersCell Biology

F. K. Jones

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

SDC3 acts as a timekeeper of myogenic differentiation by regulating the insulin/AKT/mTOR axis in muscle stem cell progeny

F. K. Jones, A. Phillips, A. R. Jones, A. Pisconti

This study identifies SDC3 as a regulator of myogenic differentiation by modulating the insulin/AKT/mTOR pathway in muscle stem cells.

bioRxiv · Aug 10, 2020 · PreprintCell Biology

MSCs from syndecan-3 null mice exhibit enhanced adhesion to collagen type I, hyperactivation of the AKT pathway and increased efficacy in inflammatory arthritis

F. K. Jones, A. Stefan, A. G. Kay, M. Hyland, R. Morgan, N. R. Forsyth, A. Pisconti, O. Kehoe

This study shows that SDC3-deficient MSCs adhere better to collagen I, have hyperactive AKT signaling, and are more effective in treating inflammatory arthritis.

bioRxiv · Jun 12, 2020Immunology

Syndecan-4-/- mice have smaller muscle fibers, increased Akt/mTOR/S6K1 and Notch/HES-1 pathways, and alterations in extracellular matrix components

S. B. Roenning, C. Carlson, J. M. Aronsen, A. Pisconti, V. Hoest, M. Lunde, K. H. Liland, I. Sjaastad, S.-O. Kolset, M. E. Pedersen

This study investigates the effects of syndecan-4 genetic ablation on muscle development and ECM remodeling in mice, finding smaller muscle fibers and altered pathway activations.

bioRxiv · Jun 10, 2020Cell Biology

Syndecan-3 enhances anabolic bone formation through WNT signalling

F. M. Johnson de Sousa Brito, A. Butcher, A. Pisconti, B. Poulet, A. Prior, G. Charlesworth, C. Sperinck, M. Scotto di Mase, G. Bou-Gharios, R. J. van 't Hof, A. Daroszewska

This study shows that Syndecan-3 promotes bone formation by enhancing WNT signaling in osteoblasts, and its absence leads to osteoporosis-like features in mice.

bioRxiv · Nov 19, 2019 · PreprintCell Biology