Author

Austin Stram

University of Wisconsin–Madison

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1 Organism:mouse· 1 Research Focus:variant calling· 1

Frequent Co-Authors

Alex C. VeithiD· 1 shared Anthony J. VeltriiD· 1 shared Christopher A. BradfieldiD· 1 shared Clifford Dustin RubinsteiniD· 1 shared Ethan Samuel LiniD· 1 shared Jae Rim WendtiD· 1 shared Jason A. CarteriD· 1 shared Jeremy D. KratziD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCancer Biology

Alex C. VeithORCID

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2 papers

Recent·Most cited

Long-read, whole-genome sequencing and chemotherapy response of two patient-derived organoids from a TP53- and KRAS-mutant ovarian carcinoma

Jae Rim Wendt, Kristin M. Adams, Ryan Moreno, Md Shahadat Hossan, Austin Stram, Ethan Samuel Lin, Lauren Kersten, Jeremy D. Kratz, Madhuchhanda Roy, Stephanie M. McGregor, Jessica D. Lang

This paper describes two patient-derived organoids from a TP53- and KRAS-mutant ovarian carcinoma, showing differential chemotherapy response and using long-read sequencing to characterize structural variants and methylation differences.

bioRxiv · Jul 10, 2026 · PreprintCancer Biology

The DNAJB1-PRKACA Oncogenic Fusion Drives Stepwise Biliary and Pancreatic Carcinogenesis from Intraductal Precursor Lesions

Patrick R. Carney, Manabu Nukaya, Jason A. Carter, Anthony J. Veltri, Kristina A. Matkowskyj, Austin Stram, Clifford Dustin Rubinstein, Alex C. Veith, Venu Gopal Pillarisetty, Christopher A. Bradfield, Sean M. Ronnekleiv‐Kelly

This study develops a mouse model of DNAJB1-PRKACA fusion and shows it drives biliary and pancreatic carcinogenesis, with pre-invasive features and Slc16a14 as a biomarker for malignant transformation.

bioRxiv · May 27, 2026 · PreprintCancer Biology