Author

B. E. Kemp

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Research Focus:metabolism & bioenergetics· 2 Assay:bulk RNA-seq· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Hoque· 1 shared A. J. Ovens· 1 shared C. G. Langendorf· 1 shared D. Yu· 1 shared J. Petersen· 1 shared J. R. C. Nunes· 1 shared J. S. Oakhill· 1 shared J. W. Scott· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperBiochemistry

A. Hoque

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Author profile →

2 papers

Recent·Most cited

Novel torin1-sensitive phosphorylation sites on the metabolic regulator AMPK revealed by label-free mass spectrometry

W. J. Smiles, A. J. Ovens, D. Yu, N. X. Y. Ling, A. Hoque, J. W. Scott, S. Galic, C. G. Langendorf, B. E. Kemp, J. Petersen, J. S. Oakhill

The paper identifies mTORC1-sensitive phosphorylation sites on AMPK using mass spectrometry, revealing their role in regulating nuclear AMPK activity and cell proliferation under nutrient stress.

bioRxiv · Oct 20, 2021 · PreprintBiochemistry

Foam cell induction activates AMPK but uncouples its regulation of autophagy and lysosomal homeostasis

N. D. LeBlond, J. R. C. Nunes, T. K. T. Smith, S. Robichaud, S. Gadde, M. Cote, B. E. Kemp, M. Ouimet, M. D. Fullerton

This study shows that AMPK activation in foam cells disrupts lysosomal homeostasis despite maintaining basal AMPK signaling, suggesting a loss of AMPK's regulatory capacity in atherosclerosis.

bioRxiv · Aug 13, 2020Cell Biology