Author

S. Galic

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Organism:mouse· 1

Frequent Co-Authors

J. S. Oakhill· 2 shared A. Hoque· 1 shared A. J. Ovens· 1 shared A. Moreno-Asso· 1 shared B. E. Kemp· 1 shared C. G. Langendorf· 1 shared C. Smith· 1 shared C.-H. Yang· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersBiochemistry

J. S. Oakhill

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Author profile →

2 papers

Recent·Most cited

Novel torin1-sensitive phosphorylation sites on the metabolic regulator AMPK revealed by label-free mass spectrometry

W. J. Smiles, A. J. Ovens, D. Yu, N. X. Y. Ling, A. Hoque, J. W. Scott, S. Galic, C. G. Langendorf, B. E. Kemp, J. Petersen, J. S. Oakhill

The paper identifies mTORC1-sensitive phosphorylation sites on AMPK using mass spectrometry, revealing their role in regulating nuclear AMPK activity and cell proliferation under nutrient stress.

bioRxiv · Oct 20, 2021 · PreprintBiochemistry

Elevated Neuropeptide Y1 Receptor Signaling Contributes to β-cell Dysfunction and Failure in Type 2 Diabetes

C.-H. Yang, D. Ann-Onda, X. Lin, S. Fynch, S. Nadarajah, E. Pappas, X. Liu, J. Scott, J. S. Oakhill, S. Galic, Y. Shi, A. Moreno-Asso, C. Smith, T. Loudovaris, I. Levinger, , K. Loh

This study shows that increased neuropeptide Y1 receptor signaling contributes to β-cell dysfunction in type 2 diabetes and that inhibiting this receptor with BIBO3304 improves β-cell function and glycaemic control.

bioRxiv · May 14, 2021 · PreprintCell Biology