Author

C. E. Seidman

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Bioengineering· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

J. G. Seidman· 2 shared A. Antolic· 1 shared D. A. Conner· 1 shared D. Y. Lee· 1 shared G. Venturini· 1 shared G. Vunjak-Novakovic· 1 shared H. Wakimoto· 1 shared J. D. Brown· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersBioengineering

J. G. Seidman

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

An engineered human cardiac tissue model reveals contributions of systemic lupus erythematosus autoantibodies to myocardial injury

S. Fleischer, T. R. Nash, M. A. Tamargo, R. I. Lock, G. Venturini, M. Morsink, V. Li, M. J. Lamberti, P. L. Graney, M. Liberman, Y. Kim, R. Z. Zhuang, J. Whitehead, R. A. Friedman, R. SONI, , G. Vunjak-Novakovic

This study uses engineered human cardiac tissues to show how SLE autoantibodies affect myocardial injury through distinct binding patterns and functional effects on cardiac cells.

bioRxiv · Mar 7, 2024Bioengineering

BET Bromodomain Proteins Regulate Transcriptional Reprogramming in Genetic Dilated Cardiomyopathy

A. Antolic, H. Wakimoto, Z. Jiao, J. M. Gorham, S. R. DePalma, D. A. Conner, D. Y. Lee, J. Qi, J. G. Seidman, J. E. Bradner, J. D. Brown, S. M. Haldar, C. E. Seidman, M. A. Burke

This study shows BET bromodomain proteins drive inflammatory gene networks in cardiac fibroblasts, contributing to genetic dilated cardiomyopathy, and that JQ1, a BET inhibitor, reduces disease progression.

bioRxiv · Feb 9, 2020Cell Biology