Author

D. Amann-Zalcenstein

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 2 Organism:human· 2 Assay:scRNA-seq· 2 Research Focus:clustering· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Organism:mouse· 1

Frequent Co-Authors

S. H. Naik· 2 shared A. P. Ng· 1 shared A. Pebay· 1 shared A. Senabouth· 1 shared A. W. Hewitt· 1 shared C. Anttila· 1 shared C. Bennett· 1 shared C. J. A. Smith-Anttila· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersCell Biology

S. H. Naik

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Author profile →

3 papers

Recent·Most cited

Spatial analysis of Hofbauer cell transcriptome, distribution and morphology in placentas exposed to Plasmodium falciparum

R. Ataide, R. Harding, M. Dharmaratne, Y. Chen, K. Fielding, L. Whitehead, K. Rogers, C. Anttila, L. Ling, P. Hickey, D. Amann-Zalcenstein, E. Moya, G. Mhango, S. Kamiza, L. Randall, , S.-R. Pasricha

The paper examines spatial transcriptomic and proteomic profiles of Hofbauer cells in placentas with and without past Plasmodium falciparum infection, finding no significant differences in HBC features but reduced fetal vessel development in infected placentas.

bioRxiv · Nov 27, 2023 · PreprintImmunology

Evolving Transcriptomic Signature of Human Pluripotent Stem Cell-Derived Retinal Pigment Epithelium Cells With Age

G. E. Lidgerwood, A. Senabouth, C. J. A. Smith-Anttila, V. Gnanasambandapillai, D. C. Kaczorowski, D. Amann-Zalcenstein, E. L. Fletcher, S. H. Naik, A. W. Hewitt, J. Powell, A. Pebay

This study characterizes the transcriptomic changes in hPSC-derived RPE cells over time in culture, showing a maturing transcriptional profile that resembles native RPE.

bioRxiv · Jul 26, 2020Cell Biology

The clonal and molecular aetiology of emergency dendritic cell development

D. S. Lin, L. Tian, S. Tomei, D. Amann-Zalcenstein, T. M. Baldwin, T. S. Weber, J. Schreuder, O. Stonehouse, J. Rautela, N. D. Huntington, A. P. Ng, S. L. Nutt, S. Taoudi, M. E. Ritchie, P. D. Hodgkin, S. H. Naik

This study investigates Flt3L-driven emergency dendritic cell development, revealing clonal expansion and molecular mechanisms in HSPCs leading to cDC1 generation.

bioRxiv · May 28, 2020 · PreprintImmunology