Author

D. Cimini

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:clinical cohort / trial· 1 Research Focus:development & morphogenesis· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

M. Bloomfield· 2 shared N. C. Baudoin· 2 shared A. L. Berr· 1 shared A. Petrosky· 1 shared B. J. Horton· 1 shared B. W. Purow· 1 shared C. K. Wong· 1 shared F. F. Bafakih· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCancer Biology

M. Bloomfield

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Whole-genome duplication shapes the aneuploidy landscape of human cancers

K. Prasad, M. Bloomfield, H. Levi, K. Keuper, S. V. Bernhard, N. C. Baudoin, G. Leor, M. Giam, C. K. Wong, G. Rancati, Z. Storchova, D. Cimini, U. Ben-David

This study shows that whole-genome duplication (WGD) alters aneuploidy patterns in human cancers, leading to distinct recurrence and genetic interaction patterns between WGD- and WGD+ tumors.

bioRxiv · May 5, 2021Cancer Biology

A patient-designed tissue-engineered model of the infiltrative glioblastoma microenvironment

R. C. Cornelison, J. X. Yuan, K. M. Tate, A. Petrosky, G. F. Beeghly, M. Bloomfield, S. R. Schwager, A. L. Berr, D. Cimini, F. F. Bafakih, J. W. Mandell, B. W. Purow, B. J. Horton, J. Munson

This study develops a patient-derived 3D tissue-engineered model of glioblastoma to assess invasion, proliferation, and stemness in response to fluid flow and glial interactions, finding CD71 expression better predicts drug efficacy than viability.

bioRxiv · Oct 2, 2020Bioengineering

Asymmetric clustering of centrosomes defines the early evolution of tetraploid cells

N. C. Baudoin, K. Soto, O. Martin, J. M. Nicholson, J. Chen, D. Cimini

The paper shows that tetraploid cells lose extra centrosomes through natural selection, favoring cells with a single centrosome in asymmetric divisions.

bioRxiv · Jan 21, 2019Cell Biology